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Combination of the cytoprotective-angiogenic component (BPC-157, VEGF/VEGFR2 pathway) with the anti-inflammatory component (KPV, NF-κB / PepT1 pathway) as a tool for the separation and comparison of mechanisms in models of mucosa repair and inflammation

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GHK peptide was also able to quench acrolein—another toxic product of lipid peroxidation involved in the development of many age-related degenerative disorders